The bongino report

How COVID Vaccines Cause Cancer

A recent study that showed IgG4 antibodies spiked in blood labs of triple-injected COVID vaccine recipients is making a lot of news.   Journalists speculate this might be why there are more cancers among the COVID-vaccinated.  The main reason COVID-vaccinated are becoming more cancerous is because of this. “turbo cancers,” Or coming out of remission after earlier cancer.  There is more evidence that earlier research has provided plausible mechanisms for cancer risk. This is based on a wealth of prior knowledge about immune function.  Let’s look at both the new study on IgG antibodies and earlier research.

These are the common fallacies: ‘Antibodies are easy to test for.  Plus, they are the focus of vaccine development and vaccine action.  So therefore we spend a lot of time thinking and talking about them.  So therefore they must be important markers of disease outcomes.  So therefore they must be decisive in disease outcomes.’

After 16 years of focusing my work as a naturopathic surgeon on cancer patients, I realized that most of my patients would have died from my misdirected efforts.

Cancer is still a problem of DNA damage, immune disruption, disrupted cells signaling, frenzied growing, lack of apoptosis and weakened tissues. These are the main features of an entity that feeds at the expense of other organs and organisms.  These are the most important features of cancer. They are difficult to treat.  This is a very daunting task. here.

First, let’s look at the new study on IgG3 versus IgG4 antibodies The triple-jabbed.  Herein, let’s call it the IgG4 study.   It found that the triple-jabbed may develop a non-inflammatory tolerance even to high levels of spike protein.  This means that instead of experiencing typical COVID symptoms like dyspnea and cough, IgG4 may be able to tolerate and even tolerize spike protein loads and allow virions to build up in the body.  A COVID+PCR result can often produce mild, sometimes even non-symptomatic, symptoms.  This could partly explain the numerous quotes from politicians and celebrities in MSM. ‘I tested positive for COVID, but thanks to my shots, it’s mild.’   Yet their lack of effective immune defeat of SARS-CoV-2 is what prevents their developing a lasting neutralizing immunity.  They are therefore able to tolerate high levels of spike protein and remain vulnerable to future infections.   Worse, the IgG4 study authors found that mild symptoms recur frequently due to a precarious immune dysfunction. This can lead to a potentially dangerous stockpiling and viral load as well as spike proteins and antibodies. This could have potentially disastrous consequences for future health outcomes.  Even a myeloma-like excess of immunoglobulins could lead to an increase in the risk of developing a multiple myeloma-like disease The COVID-vaccinated have a high-protein, low-quality blood that can cause damage to the kidneys’ fine filtration systems, the glomeruli.

As immune deviation, misdirection and disorder have been described previously Pathogenic primingMaladaptation is when the immune system tries to avoid or ineffectively fight real threats and focuses its resources to defeat non-threatening ones.  This was evident in the development of the mRNA vaccinations that produced a spike protein, which was typical of the original Wuhan strain SARS-CoV-2. However, it proved ineffective against Omicron, Delta and other strains. warned.  The COVID vaccinations were not available to the public because the Wuhan strain had already been emitted.

Natural infection can cause IgM antibodies to flare up for a brief time. IgG antibodies are slower to develop and last longer after the infection has gone away.  For example, IgGs for measles are still strong in a blood laboratory decades after I got measles as an infant. This is because I have only natural immunity and no vaccine history.

IgG4 is a subclass that is non-inflammatory and is related to tolerance to antigens.   IgG4 does not have an effector function.  IgG4 appears to be ininversely related with anaphylaxis.   In the IgG4 paper, the IgG4 levels for COVID-vaccinated individuals are shown to increase 38-fold after the third mRNA injection.  The logarithmic scale of the y axis places the IgG4s very high up.

The triple- and double jabbed simultaneously lose a significant amount of IgG3 antibodies. This was discovered at 180-day follow up labs and 210 day follow-up labs.  You can see the logarithmic scale showing the dramatic drop-off IgG3 antibody levels and skyrocketing IgG4 antibody levels.   This is Figure 1. IgG4 paper:

Epoch Times Photo

Sometimes, the subclass IgG3 has been thought by IgG4 authors to be pro-inflammatory. It is one of many immune attacks against invading pathogens.  Although there are no known anti-inflammatory subclasses, it is believed to be pro-inflammatory. evidence to the contrary Also.  IgG3 is sometimes used by IgG4 authors and other interested journalists to fight or neutralize antigens.

There is no evidence to support the idea that IgG3 antibodies are effective in fighting pathogens, except for correlation of titers.  The IgG4 authors acknowledge an earlier observation. “IgG3 responses correlating with partial protection against HIV,” Only a slight increase in IgG3 antibody levels after SARS-CoV-2 natural infection was reported. hereWithout protection mechanism.

One possible clue as to the IgG4 study authors’ observations of low IgG3 is the glycosylation of IgG3 SARS-CoV-2 severity is affected.  (Immunoglobulins, which are generally glycosylated protein molecules, seem to have hyper-glycosylation as a problem.  Glycosylation can be detrimental to the optimal function of an immune system; in our junk food-loving culture, glycosylation has caused more damage than just antibodies.

IgG3 antibodies make up a small percentage of IgG antibodies. have not yet been well-studied.  IgG3 and IgG4 antibodies represent a small portion of all our B cells, approximately 3% and 4%, respectively.

Low IgG3 antibodies do not always correlate with low disease severity.  COPD, for example, is a case where we see this correlation This is Low IgG3 levels in patients with COPD that are experiencing life-threatening symptoms.  All antibodies including IgG3 and IgG4In natural infection, the IgG3/IgG4 ratios generally rise and then drop.  I will now explain why I doubt the cause and effects vector works as assumed. It could range from low IgG3/IgG4 to generalized immune dysfunction.  It may be more likely to be an effect of other mechanisms as described below.

First, the IgG fascination has the problem of assuming that because antibodies are easy to measure and therefore have a large impact on the complex immune system, they must be important.  Metaphorically speaking, we are assuming that what we can see is decisive. In other words, we are seeing the skin and assume that the function of the internal organs is known and the skin is the predominant cause of these internal effects.  Evidently, this is not the case.

Let’s first assume that the highly mobile and ubiquitous blood contains many of the cells in our immune system and are, as a whole and in parts, key to optimal immune function.  Here is how much IgG immunoglobulin antibodies are in relation to the rest.

The surface of B-cells is home to immunoglobulins. They act as receptors for certain antigens.  While their number can fluctuate, they make up 5.2% to 5.1% of all white blood cells.  White blood cells account for 0.1% in all blood cells.  Therefore, B-cells make up 0.00005% of all blood cells or 5 out 100,000 cells.

I will explain more about this. here.

Epoch Times Photo

The proportion of blood-cells in B-cells is tiny.  You can see the thin red line at the bottom of the image below. This is the percentage of all the B-cells relative to the vast majority of blood cells.  (The thin red line at the far left of the band below would have to be thinner in order to be true to scale.

Epoch Times Photo

Now let’s look at other aspects of immune function that are powerhouse fighters against cancer, but have been associated with high viral load and/or high spike protein, such as is expected to occur after COVID vaccination.  The researchers discovered that the two most important cancer-fighting cells in our bodies are natural killers. (NK) cells and CD8+ T-cells were significantly reduced These are the circumstances.  For more aggressive tumors, a decrease in NK cell count is common.

The major problem with the mRNA vaccines COVID and the cancer risk was revealed in April this year. Seneff, Nigh paper.

The science community’s pre-occupation with the relatively smaller adaptive immune system, mostly its humoral portion, and unfamiliarity or disinterest in the vastly more important and stronger innate immune system has led attention away from this seminal paper.   The Seneff-Nigh paper is a must-read. It provides the best information available on the impact of COVID vaccinations on tumorigenesis, immune failure with respect to cancer, and metastatic events.

Seneff and colleagues found that the greatest threat to immune function from mRNA vaccines was the interferon signaling pathways.  This affects the immune system’s ability to detect cancer.  In COVID-vaccinated patients, we often see metastases and new tumors.  Turbo cancers are now common.  This is how Seneff and colleagues support that hypothesis.  The paper is very detailed and I will summarize it below.

Ivanova, et al It was found that those who had been naturally infected by SARS-CoV-2 were able to significantly increase the levels of our most important cytokine, Type 1 interferon. This ability was not seen in mRNA-vaccinated individuals.  It is clear that COVID vaccines reduce Type I interferon signalsing, based on these findings.  This results in a severe breakdown of many downstream immune systems, making people more susceptible to both viral and cancerous diseases. The necessity of interferons for the body’s war against cancer is further seen in the productive clinical use over decades of interferon as a therapeutic agent to cancer patients.

The most appreciated mechanisms of Type I interferon against cancers include up-regulation of the tumor suppressor gene p53, as well as kinase inhibitors, and the resulting arrest of cancer’s cell reproduction.  Interferon-alpha is a type 1 interferon. makes cancer recognizableOr in a manner that is visible to other immune cell for destruction.  Type I interferons, particularly interferon alpha, also have major effects on cell differentiation and death. These are two major events that are essential for natural victories over cancer.  Type I interferon activates CD8+ and the NK cells, which are essential for fighting cancer.  Type I interferons also have genetic effects that suppress tumors. IRF-7 genes.  These genes have an effect on cancers The breast, prostate and uterus as well as the ovaries, pancreas, and uterus.  These and other oncogenes are generally dysregulated by mRNA vaccines.

Fay et al G-quadruplex formations are found in RNA. This is also discussed in relation to proto-oncogene expression.  This could lead to the development of cancer.

The Vaccine Adverse Events Reporting System of the Dept of Health and Human Services showed that the number of cancers caused by COVID vaccines was higher than for any other vaccines over the 30-year-old history of VAERS.  These new cancers, which were caused by the COVID vaccinations, accounted for 98%.  Seneff and colleagues again:

Epoch Times Photo

It should be noted that the reporting of these 2021 cancers occurred in large part prior to the US public’s (tepid) uptake of even the earliest mRNA COVID boosters (injection #3 in the fall of 2021) as here shown in Our World in Data.

Epoch Times Photo

The 3rd injection is when the IgG4 paper author saw the largest difference in IgG3/IgG4 proportions but not necessarily the greatest rise in cancer cases.

Let’s consider the whole immune system, not only immunoglobulins, as necessary to protect against the ravages of cancer.  The irreversible injections of new products such as the mRNA vaccinations must be stopped from destroying immune cells and cytokines.

Reposted from the author’s Substack


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